Smells LIKE EGGS!
My first guess would've been the cat as well. Those thing smell awful when they burn out. But if you are sure it’s not that, and found a melted hose, I would think its burning transmission fluid. The stuff already smells bad, and when it gets burned it really reeks. Maybe that was a vent line or cooler line that got melted and fluid it coming out?? There wasn’t any smoke coming out from under the Jeep was there?
That's what I thought.
Mine is the manual transmission and during the first few times I backed up my driveway (it is steep), I notice it a little.
Doesn't happen anymore though.
I was going to guess clutch, but there are some other good theories here too
(I'll pass on the walking farts) wait.. that didnt come out right..
neither did that. GAH!
Just thinking about what happened and maybe that Fuc$%#g mechanic did it on purpose because I TOLD him to take the hood off the windshield.....
Here's a couple pics of what I am talking about...notice the new rubber they put as a temp solution??
Here's a couple pics of what I am talking about...notice the new rubber they put as a temp solution??
Hydrogen Sulfide is very toxic...
"Toxicity
Hydrogen sulfide is considered a broad-spectrum poison, meaning that it can poison several different systems in the body, although the nervous system is most affected. The toxicity of H2S is comparable with that of hydrogen cyanide. It forms a complex bond with iron in the mitochondrial cytochrome enzymes, thereby blocking oxygen from binding and stopping cellular respiration. Since hydrogen sulfide occurs naturally in the environment and the gut, enzymes exist in the body capable of detoxifying it by oxidation to (harmless) sulfate. Hence low levels of sulfide may be tolerated indefinitely. However, at some threshold level, the oxidative enzymes will be overwhelmed. This threshold level is believed to average around 300-350 ppm. Many personal safety gas detectors are set to alarm at 10 PPM and to go into high alarm at 15 PPM (Utility, sewage & petrochemical workers).
An interesting diagnostic clue of extreme poisoning by H2S is the discoloration of copper coins in the pockets of the victim. Treatment involves immediate inhalation of amyl nitrite, injections of sodium nitrite, inhalation of pure oxygen, administration of bronchodilators to overcome eventual bronchospasm, and in some cases hyperbaric oxygen therapy.
Exposure to lower concentrations can result in eye irritation, a sore throat and cough, shortness of breath, and fluid in the lungs. These symptoms usually go away in a few weeks. Long-term, low-level exposure may result in fatigue, loss of appetite, headaches, irritability, poor memory, and dizziness. Chronic exposures to low level H2S (around 2ppm) has been implicated in increased miscarriage and reproductive health issues amongst Russian and Finnish wood pulp workers, but the reports hadn't (as of circa 1995) been replicated. Higher concentrations of 700-800 ppm tend to be fatal.
* 0.0047 ppm is the recognition threshold, the concentration at which 50% of humans can detect the characteristic odor of hydrogen sulfide [1], normally described as resembling "a rotten egg".
* 10-20 ppm is the borderline concentration for eye irritation.
* 50-100 ppm leads to eye damage.
* At 150-250 ppm the olfactory nerve is paralyzed after a few inhalations, and the sense of smell disappears, often together with awareness of danger,
* 320-530 ppm leads to pulmonary edema with the possibility of death.
* 530-1000 ppm causes strong stimulation of the central nervous system and rapid breathing, leading to loss of breathing;
o 800 ppm is the lethal concentration for 50% of humans for 5 minutes exposure(LC50).
* Concentrations over 1000 ppm cause immediate collapse with loss of breathing, even after inhalation of a single breath.
A practical test used in the oilfield industry to determine whether someone requires overnight observation for pulmonary edema is the knee test: if a worker that gets "gassed" loses his balance and at least one knee touches the ground, the dose was high enough to cause pulmonary edema."
"Toxicity
Hydrogen sulfide is considered a broad-spectrum poison, meaning that it can poison several different systems in the body, although the nervous system is most affected. The toxicity of H2S is comparable with that of hydrogen cyanide. It forms a complex bond with iron in the mitochondrial cytochrome enzymes, thereby blocking oxygen from binding and stopping cellular respiration. Since hydrogen sulfide occurs naturally in the environment and the gut, enzymes exist in the body capable of detoxifying it by oxidation to (harmless) sulfate. Hence low levels of sulfide may be tolerated indefinitely. However, at some threshold level, the oxidative enzymes will be overwhelmed. This threshold level is believed to average around 300-350 ppm. Many personal safety gas detectors are set to alarm at 10 PPM and to go into high alarm at 15 PPM (Utility, sewage & petrochemical workers).
An interesting diagnostic clue of extreme poisoning by H2S is the discoloration of copper coins in the pockets of the victim. Treatment involves immediate inhalation of amyl nitrite, injections of sodium nitrite, inhalation of pure oxygen, administration of bronchodilators to overcome eventual bronchospasm, and in some cases hyperbaric oxygen therapy.
Exposure to lower concentrations can result in eye irritation, a sore throat and cough, shortness of breath, and fluid in the lungs. These symptoms usually go away in a few weeks. Long-term, low-level exposure may result in fatigue, loss of appetite, headaches, irritability, poor memory, and dizziness. Chronic exposures to low level H2S (around 2ppm) has been implicated in increased miscarriage and reproductive health issues amongst Russian and Finnish wood pulp workers, but the reports hadn't (as of circa 1995) been replicated. Higher concentrations of 700-800 ppm tend to be fatal.
* 0.0047 ppm is the recognition threshold, the concentration at which 50% of humans can detect the characteristic odor of hydrogen sulfide [1], normally described as resembling "a rotten egg".
* 10-20 ppm is the borderline concentration for eye irritation.
* 50-100 ppm leads to eye damage.
* At 150-250 ppm the olfactory nerve is paralyzed after a few inhalations, and the sense of smell disappears, often together with awareness of danger,
* 320-530 ppm leads to pulmonary edema with the possibility of death.
* 530-1000 ppm causes strong stimulation of the central nervous system and rapid breathing, leading to loss of breathing;
o 800 ppm is the lethal concentration for 50% of humans for 5 minutes exposure(LC50).
* Concentrations over 1000 ppm cause immediate collapse with loss of breathing, even after inhalation of a single breath.
A practical test used in the oilfield industry to determine whether someone requires overnight observation for pulmonary edema is the knee test: if a worker that gets "gassed" loses his balance and at least one knee touches the ground, the dose was high enough to cause pulmonary edema."
Have you been filling up with premium gas? If you have, try regular. Some vehicles require regular grade gas, and if you run premium it can smell like rotten eggs. My chevy c3500 with 350ci was like that. I changed to regular and the smell went away...


